Hashimoto et al. Bilateral hypogeusia caused by a small lesion in the lower midbrain tegmentum. BMJ Case Rep. 2012 Sep 11;2012. pii: bcr2012006837. doi: 10.1136/bcr-2012-006837.
This is a case report of an MSer who developed loss of taste on both sides of the tongue and mouth (bilateral hypogeusia) due to a small lesion in the lower midbrain tegmentum (area in the brainstem). Sweet taste was diminished only on one side and salty, sour and bitter tastes were diminished bilaterally.
This is a case report of an MSer who developed loss of taste on both sides of the tongue and mouth (bilateral hypogeusia) due to a small lesion in the lower midbrain tegmentum (area in the brainstem). Sweet taste was diminished only on one side and salty, sour and bitter tastes were diminished bilaterally.
“Loss of taste is actually quite rare in MS, but we don’t know how rare! Let’s find out?”
“Have any of you had a relapse that has affected your taste? Please complete the survey below!”
ive had it but didn't realise it was a rare symptom. was accompanied by numbness in my cheek and weaker than usual speech.
Yes my taste buds were altered when I had a relapse that seemed to affect my jaw, I thought it was my wisdom teeth playing up, everything tasted less intense, don't think they came back 100% to what they were, just got used to it. My speech was slightly slurred as well at the time.
I'm posting this here because i could not post it in the entry "Research: CCSVI papers back after summer holiday".You did miss some of the papers published (related to CCSVI), so i post them here if you want to discuss them and give some insight about what they say:"Iron content of the pulvinar nucleus of the thalamus is increased in adolescent multiple sclerosis."http://www.ncbi.nlm.nih.gov/pubmed/22968543Cerebral vein changes in relapsing-remitting multiple sclerosis demonstrated by three-dimensional enhanced T (2) (*) -weighted angiography at 3.0 T.http://www.ncbi.nlm.nih.gov/pubmed/22968782
Dear Anon 3:45Maybe read the blog I think you are missing things.with regard http://www.ncbi.nlm.nih.gov/pubmed/22968782. If you look back to 13 september and you will see this post and with regard to http://www.ncbi.nlm.nih.gov/pubmed/22968543. We do not do a post on every paper that is published and had no plans on posting"The objective of the paper is to assess abnormal phase values, indicative of increased iron content", using susceptibility-weighted imaging (SWI)-filtered phase of the subcortical deep gray matter (SDGM) in adolescent multiple sclerosis (MS) and other neurological disorders (OND) patients, and in healthy controls (HC)". "CONCLUSION: Adolescent MS patients showed increased iron content in the SDGM compared to OND patients and HC".Whilst Iron deposition is part of the Zamboni hypothesis, it is accepted that iron is important for brain function, this is why blood vessels have iron transporters (transferrin receptors) in them to pump iron into the brain. Likewise it is probably accepted that there are changes in iron biology in MS. This paper reports that there may be changes in young MSers, but if iron changes are evident in MS, So why report on this? It is nothing new and it is neither here nor there with regard to validity of CCSVI and can this MRI outcome really detect what it claims to detect. I didn't find it interesting for me to report, but I am not an MRIer. Maybe if Dr Klaus would post you may get exciting MRI posts.
why not post in unrelated comments spot if it does not fit with the thread
I've had two separate instances of a drastic reduction in taste, rating at about a 3/5 (from two separate exacerbations). I suspect I may have a related lesion as I have also experienced burning/tingling in the middle of my tongue constantly for the last 1.5 years (before I was diagnosed and straight through relapse and remission). I'm a female in my mid thirties. Oddly, acidic or salty foods seem to make burning/tingling more intense but avoiding them doesn't make it go away either.